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Important: Please avoid administering Pfizer or Moderna on our kids, teens & NSFs

VAX%20PUSHBACK%20(1).webp
 
In a recently published paper published in Biomedicines titled, “‘Spikeopathy’: COVID-19 Spike Protein Is Pathogenic, from Both Virus and Vaccine mRNA,” the researchers explored peer-reviewed data countering the “safe and effective” narrative attached to new technologies used to develop mRNA and adenovectorDNA vaccines at “warp speed” to end the pandemic.

Spike protein pathogenicity, termed “spikeopathy,” describes the ability of the spike protein to cause disease, and the researchers say it can affect many organ systems. Researchers noted the following key problem areas:

  • Spike protein toxicity (spikeopathy) from both the virus and when produced by gene codes in people vaccinated with COVID-19 vaccines.
  • Inflammatory properties in specific lipid nanoparticles (LNPs) used to transport mRNA.
  • Long-lasting action caused by N1-methyl pseudouridine in the synthetic mRNA—also referred to as modRNA.
  • Widespread distribution of mRNA and DNA codes via the LNP and viral vector carrier matrices, respectively.
  • Human cells produce a foreign protein that can cause autoimmunity.
Now that vaccines utilizing mRNA technology have been available and widely distributed for several years, data show these vaccines produce foreign antigens in human tissues and increase the risk of autoimmune, neurological, cardiovascular, inflammatory disorders, and cancers, especially when the vaccine ingredients do not remain localized at the injection site. An antigen is any substance that stimulates an immune response. If the immune system encounters an antigen that is not found on the body’s own cells, it will launch an attack against that antigen.

Pharmacokinetic and pharmacodynamic data show the design of the mRNA and adenovectorDNA COVID-19 vaccines allow uncontrolled biodistribution, durability, and persistent bioavailability of the spike protein inside the body after vaccination. Pharmacokinetics is the study of how the body interacts with administered substances for the entire duration of exposure. Pharmacodynamics assesses the drug’s effect on the body more closely.

This may explain the unprecedented number of adverse events that appear to be associated with the spike protein produced by the gene-based technologies employed by Pfizer, Moderna, AstraZeneca, and Johnson & Johnson, as well as the viral vector DNA technology used by other countries, researchers said.

https://www.mdpi.com/2227-9059/11/8/2287
Published: 17 August 2023
 
These adverse effects already surfaced in the Gardasil mRNA vaccines.

Yet, BIG pharma just ignored this fact.

In the US vaccine court, there are cases in which the victims sued for damages, and won. A little known fact.
 
It is worrying, SOS lost count, will this the fifth or sixth jab?

even the professors at NUS Saw Swee Hock School Of Public Health have their hesitations or at least, not completed their reviews because it is insufficiently-tested or has limited data. Now we just get some GPs to speak for the medical community?????????


Doctors reiterate importance of vaccination and boosters amid recent spike in COVID-19 cases
https://www.channelnewsasia.com/sin...ne-booster-importance-upgraded-pfizer-3779061

SINGAPORE: Doctors are reiterating the importance of staying up-to-date with COVID-19 vaccinations amid a rise in cases. General practitioners (GPs) told CNA they have seen a surge in patients in recent weeks, including cases of reinfection.
 
Come late October, I expect the pro-vaccine propaganda machinery to work its magic again. Look for interviews of enthusiastic jab addicts, most likely the old farts, to camp at JVTCs and give their 'approved' 2-minute soundbites to those so-called journalists. :wink:




 
SOS said:
It is worrying, SOS lost count, will this the fifth or sixth jab?

even the professors at NUS Saw Swee Hock School Of Public Health have their hesitations or at least, not completed their reviews because it is insufficiently-tested or has limited data. Now we just get some GPs to speak for the medical community?????????


Doctors reiterate importance of vaccination and boosters amid recent spike in COVID-19 cases
https://www.channelnewsasia.com/sin...ne-booster-importance-upgraded-pfizer-3779061

SINGAPORE: Doctors are reiterating the importance of staying up-to-date with COVID-19 vaccinations amid a rise in cases. General practitioners (GPs) told CNA they have seen a surge in patients in recent weeks, including cases of reinfection.
Click to expand...

The only common thing between unvaxxed purebloods and the vaxtards is that none of us will ever be 'fully vaccinated'. :cool:
 
I'll post here what I posted in Sam's thread "Concidence?"

* * *
In a nutshell, this is how an mRNA vaccine works:
1. The mRNA is injected, enters the bloodstream, and circulates round the body
2. It is then taken up by the cells in the lining of the blood vessels at various sites
3. The cells make use of this mRNA (coded genetic material) to produce a spike protein similar to the COVID-19 virus spike protein
4. This spike protein then triggers the body's immune system to form antibodies against it, giving immunity.

Three crucial factors you can't control with the mRNA approach:
1. There's absolutely no way you can determine which organs or tissues take up the mRNA as it circulates in the bloodstream
2. There's absolutely no way to control how much mRNA is taken up into the cells at a particular site
3. There's absolutely no way to control how much spike protein is produced by the cell once the mRNA enters it

It has been shown that in people susceptible to the deleterious effects of the mRNA vaccine tend to take up more mRNA and produce a lot more of the spike protein in the their heart and brain vessels -> triggering an excess of antibodies that damage these vessels (auto-immune damage) -> micro-clots -> stroke, heart attack, inflammation of heart muscle.

Inactivated Virus Vaccine (Sinopharm, Sinovac)
This is old technology, like the flu vaccine. Take some virus, inactivate it, inject it into the body, let the dead virus circulate and stimulate the immune system. Optimal dosage determined in trials.

In other words, you're attempting to give the person a very, very mild 'infection', but without the virus multiplying and causing more severe symptoms.

Yes, inactivated virus vaccines stimulate a weaker response (in terms of antibody levels) than mRNA vaccines, but they prevent severe COVID-19 illness equally effectively. And most importantly: they're far safer.

To date, there's been no report of severe adverse events with inactivated virus vaccines locally save for allergic reactions, which can occur with any drug or vaccine.

I'd rather have a slightly less effective but safer vaccine than a more effective but highly dangerous vaccine. Remember, even with a quoted incidence of 1 in 10,000 cases for severe adverse events in mRNA vaccine use (a BS figure conjured up by Big Pharma and their captive governments), for the victim who dies from a heart attack it's a 100% fatality rate.

Your mileage, as always, may differ. (And you'll never read about the above in the Stooge Times or Western media.)
 
Wuhan Institute of Biology is a Biosafety Level 4 lab funded by Fauci and the US NIH to do 'gain of function' studies on various viruses, including the coronavirus. These studies basically tweak viruses to make them more virulent for the purpose of developing newer treatments and vaccines to pre-empt future pandemics, as well as creating bioweapons.

Anyone with half a brain knows that the SARS-Cov-2 virus was accidentally leaked from the lab, spread to the market nearby, and the rest is history.

The Chinese and Americans have blood on their hands. They're both complicit in the cover-up.
 
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Why Scientists Tweak Lab Viruses to Make Them More Contagious​

Some “gain of function” studies explore how a dangerous pathogen might cross species barriers to start an outbreak. They are not without controversy
The microbiology toolbox includes techniques to induce mutations in viruses that give the microbes new powers. Scientists perform these manipulations for many reasons, including wanting to understand how the microbes evade detection by our immune systems. But adding capability to a pathogen carries obvious risks, especially if this “gain of function” involves enhanced virulence or infectiousness. Escape from a lab, by accident or design, is a possibility. So why do it? Some researchers argue the work can offer a peek at what a virus can do before it goes into the natural world and poses a threat to people.

Controversy over gain-of-function research has generated academic papers, conferences and even a moratorium in 2014, when the U.S. government paused funding for three years until steps could be taken to ensure the safety of the procedure. Debate about gain-of-function experiments continues in the latter phases of the pandemic as thoughts turn to the “next one” or a possible second act for COVID-19. Science policy makers must wrestle with defining the rare instances in which the benefits of experiments that enhance a virus’s capacity to survive and flourish in human hosts outweigh any risks.

Densely technical discussions often bog down over the very definition of gain of function. Recently, semantics were front and center in the debate over whether National Institutes of Health–funded work at the Wuhan Institute of Virology (WIV) in China constituted gain-of-function research, a contention denied by the U.S. agency. The WIV has also been the focus of a revived dispute over whether SARS-CoV-2, the virus that causes COVID-19, escaped from its facility.

Here are a few basic answers to questions about why an obscure technical term now receives so much attention.

What is gain of function research?

Techniques to enhance some aspect of an organism’s functioning are commonplace in research and applied to everything from mice to measles. One typical application of this approach is tweaking mouse genes to generate more of a protein that limits fat deposition.

But that is not the kind of gain-of-function study that raises fears among scientists and regulators. The high-risk practices are those that create mutations to examine whether a pathogen becomes more contagious or lethal as a means of estimating future threats.

Some experts acknowledge the critical differences between the two types of studies. One proposed term to represent the more threatening subset of this research is “potential pandemic pathogens,” says Marc Lipsitch, a professor of epidemiology at the Harvard T. H. Chan School of Public Health. That phrase “singles out the name and reason for being concerned,” he adds. It has not caught on in common usage, however, returning only about 8,500 results in a Google search, compared with 13.4 million for “gain of function.”

Making this distinction is important for a few reasons, Lipsitch says. When the U.S. government placed the 2014 moratorium on “gain of function research,” some of the studies that were affected carried no obvious risk of setting off a pandemic.

What is the purpose of this research?

Knowing what makes a microbe more dangerous enables preparation of countermeasures, says Lipsitch, who is one of 18 signatories to a May 14 letter, published in Science, that calls for the investigation of a SARS-CoV-2 lab spillover as one of several possible explanations for the origins of the COVID-19 pandemic. He points to the difficulties of studying viruses for the development of vaccines and treatments without doing experiments in a mouse or in other nonhuman animals. There is, Lipsitch says, a “direct path from doing that research to gaining public health benefits,” enabling a balancing of risks and potential benefits.

The riskier version of gain-of-function research creates viruses with abilities they do not have in nature. In two separate studies in 2011, scientists famously and controversially did just that with the H5N1 influenza virus, or “bird flu,” resulting in a version capable of airborne transmission among ferrets. The naturally occurring virus does not have this ability. Making mammal-to-mammal transmission easier set off alarm bells and triggered discussion of a U.S. moratorium.

In 2015 researchers engineered a hybrid pathogen that combined features of the original SARS virus (SARS-CoV) that infected humans in the early 2000s with that of a bat coronavirus. Most bat coronaviruses cannot infect the cells lining the human respiratory tract. This experiment was intended to mimic what would happen if a third species served as a mixing vat for the bat and human viruses to exchange genetic material. The result was a pathogen that could enter human cells and also cause disease in mice. Reactions to this work were polarized, as demonstrated by experts quoted in a 2015 article in Nature: one said that all the research did was create a “new, non-natural risk” among the multitude that already exist, while another contended that it showed the potential for this bat virus to become a “clear and present danger.”

https://www.scientificamerican.com/...ak-lab-viruses-to-make-them-more-contagious1/
 
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