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why pap still allowing motherfucking ceca family members to return to singapore and spread covid?

kaninabuchaojibye

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knn right!
why should we as singaporeans pay the price of their stupid decision to allow these ceca families into singapore when vivid is raging in india?

nabeipuacheebye PAP
 
those who allowed these hordes in, you know who you are, hope you all get stroke and cant move for 50 years, knnccb
 
Lawlan already said the Covid-19 task force have done a good job....
 
CB laulan , all foreigners come back here for free treatment n lodging , CB
 
It is pretty much established, based upon data from Sweden, NYC and many other locations which had high case counts, that herd immunity is not 70% but is actually far lower viz in the 20% range.

The reason for this is that 80% of all populations are immune to Covid-19.

This immunity comes from the fact that many of us have already been exposed to coronavirus family of viruses and this apparently impart T cell immunity even though antibody testing is negative.

The best way of protecting the vulnerable ie those with pre existing conditions and the elderly is to allow the virus to spread through the healthy population. There will be some pain but nothing worse than seasonal flu.

Once the virus has run out of people to infect the pandemic is over. The vulnerable can then emerge from the bunkers.
 
Government said we need these foreign PMET, they create better and higher pay jobs for us, otherwise we have no jobs, no investors would come to Singapore
 
Lawlan already said the Covid-19 task force have done a good job....
The mistake they have made has been to impose a hard lockdown (circuit breaker) instead of allowing a slow spread of the virus.

The aim was to not overwhelm the health system. It was never about achieving zero cases to begin with. For some strange reason the narrative changed midway.
 
https://en.wikipedia.org/wiki/Common_cold

Viruses



Coronaviruses are a group of viruses known for causing the common cold. They have a halo or crown-like (corona) appearance when viewed under an electron microscope.

The common cold is a viral infection of the upper respiratory tract. The most commonly implicated virus is a rhinovirus (30–80%), a type of picornavirus with 99 known serotypes.[29][30] Other commonly implicated viruses include human coronaviruses (≈ 15%),[31][32] influenza viruses (10–15%),[33] adenoviruses (5%),[33] human respiratory syncytial virus (orthopneumovirus), enteroviruses other than rhinoviruses, human parainfluenza viruses, and human metapneumovirus.[34] Frequently more than one virus is present.[35] In total, more than 200 viral types are associated with colds.[3]
 
Infection in humans



Illustration of SARSr-CoV virion

Coronaviruses vary significantly in risk factor. Some can kill more than 30% of those infected, such as MERS-CoV, and some are relatively harmless, such as the common cold.[43] Coronaviruses can cause colds with major symptoms, such as fever, and a sore throat from swollen adenoids.[81] Coronaviruses can cause pneumonia (either direct viral pneumonia or secondary bacterial pneumonia) and bronchitis (either direct viral bronchitis or secondary bacterial bronchitis).[82] The human coronavirus discovered in 2003, SARS-CoV, which causes severe acute respiratory syndrome (SARS), has a unique pathogenesis because it causes both upper and lower respiratory tract infections.[82]

Six species of human coronaviruses are known, with one species subdivided into two different strains, making seven strains of human coronaviruses altogether.




Seasonal distribution of HCoV-NL63 in Germany shows a preferential detection from November to March

Four human coronaviruses produce symptoms that are generally mild:


  1. Human coronavirus OC43 (HCoV-OC43), β-CoV
  2. Human coronavirus HKU1 (HCoV-HKU1), β-CoV
  3. Human coronavirus 229E (HCoV-229E), α-CoV
  4. Human coronavirus NL63 (HCoV-NL63), α-CoV

Three human coronaviruses produce symptoms that are potentially severe:


  1. Middle East respiratory syndrome-related coronavirus (MERS-CoV), β-CoV
  2. Severe acute respiratory syndrome coronavirus (SARS-CoV), β-CoV
  3. Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), β-CoV
Common cold

Main article: Common cold

The human coronaviruses HCoV-OC43, HCoV-HKU1, HCoV-229E, and HCoV-NL63 continually circulate in the human population and produce the generally mild symptoms of the common cold in adults and children worldwide.[83] These coronaviruses cause about 15% of common colds,[84] while 40 to 50% of colds are caused by rhinoviruses.[85] The four mild coronaviruses have a seasonal incidence occurring in the winter months in temperate climates.[86][87] There is no preponderance in any season in tropical climates.[88]
 
Lanjiao Loong needs to catch covid from these filthy shitskins and have his corpse whipped!
 
The mistake they have made has been to impose a hard lockdown (circuit breaker) instead of allowing a slow spread of the virus.

The aim was to not overwhelm the health system. It was never about achieving zero cases to begin with. For some strange reason the narrative changed midway.
The banglas knew about this and successfully implemented "swedish method" without any inteference and have very few casualties despite the high numbers of infection.
The despicable local chinese immigrant populace trying to distract attention from the fact that its a china virus and putttig the blame on banglas for the disease.
 
The mistake they have made has been to impose a hard lockdown (circuit breaker) instead of allowing a slow spread of the virus.

The aim was to not overwhelm the health system. It was never about achieving zero cases to begin with. For some strange reason the narrative changed midway.

Chan needed a good crisis to save the party :wink:
 
It is pretty much established, based upon data from Sweden, NYC and many other locations which had high case counts, that herd immunity is not 70% but is actually far lower viz in the 20% range.

The reason for this is that 80% of all populations are immune to Covid-19.

This immunity comes from the fact that many of us have already been exposed to coronavirus family of viruses and this apparently impart T cell immunity even though antibody testing is negative.

The best way of protecting the vulnerable ie those with pre existing conditions and the elderly is to allow the virus to spread through the healthy population. There will be some pain but nothing worse than seasonal flu.

Once the virus has run out of people to infect the pandemic is over. The vulnerable can then emerge from the bunkers.
Herpe can also get immunity? :unsure:
 

This is the figure for HSV1 alone.

who.int

Globally, an estimated two-thirds of the population under 50 are infected with herpes simplex virus type 1


5-6 minutes


More than 3.7 billion people under the age of 50 – or 67% of the population – are infected with herpes simplex virus type 1 (HSV-1), according to WHO’s first global estimates of HSV-1 infection published today in the journal PLOS ONE.
Herpes simplex virus is categorized into 2 types: herpes simplex virus type 1 (HSV-1) and herpes simplex virus type 2 (HSV-2). Both HSV-1 and HSV-2 are highly infectious and incurable. HSV-1 is primarily transmitted by oral-oral contact and in most cases causes orolabial herpes or “cold sores” around the mouth. HSV-2 is almost entirely sexually transmitted through skin-to-skin contact, causing genital herpes.
“Access to education and information on both types of herpes and sexually transmitted infections is critical to protect young people’s health before they become sexually active,”
Dr Marleen Temmerman, Director of WHO’s Department of Reproductive Health and Research
The new estimates highlight, however, that HSV-1 is also an important cause of genital herpes. Some 140 million people aged 15-49 years are infected with genital HSV-1 infection, primarily in the Americas, Europe and Western Pacific. Fewer people in high-income countries are becoming infected with HSV-1 as children, likely due to better hygiene and living conditions, and are instead at risk of contracting it genitally through oral sex after they become sexually active.
“Access to education and information on both types of herpes and sexually transmitted infections is critical to protect young people’s health before they become sexually active,” says Dr Marleen Temmerman, Director of WHO’s Department of Reproductive Health and Research.
Large number of genital infections
In January, WHO estimated that 417 million people aged 15-49 years have HSV-2 infection, which causes genital herpes. Taken together, the estimates reveal that over half a billion people between the ages of 15-49 years have genital infection caused by either HSV-1 or HSV-2.
“The new estimates highlight the crucial need for countries to improve data collection for both HSV types and sexually transmitted infections in general,” says Dr Temmerman.
Given the lack of a permanent and curative treatment for both HSV-1 and HSV-2, WHO and partners are working to accelerate development of HSV vaccines and topical microbicides, which will have a crucial role in preventing these infections in the future. Several candidate vaccines and microbicides are currently being studied.
Regional infection estimates
Estimates for HSV-1 prevalence by region among people aged 0-49 in 2012:
  • Americas: 178 million women (49%), 142 million men (39%)
  • Africa: 350 million women (87%), 355 million men (87%)
  • Eastern Mediterranean: 188 million women (75%), 202 million men (75%)
  • Europe: 207 million women (69%), 187 million men (61%)
  • South-East Asia: 432 million women (59%), 458 million men (58%)
  • Western Pacific: 488 million women (74%), 521 million men (73%)
Estimates of new HSV-1 infections among people aged 0-49 in 2012:
  • Americas: 6 million women, 5 million men
  • Africa: 17 million women, 18 million men
  • Eastern Mediterranean: 6 million women, 7 million men
  • Europe: 5 million women, 5 million men
  • South-East Asia: 13 million women, 14 million men
  • Western Pacific: 11 million women, 12 million men
Disease symptoms
Herpes is a lifelong infection, which often has mild or no symptoms but can be detected by the presence of antibodies for HSV-1 or HSV-2 in the blood. It is difficult to determine the proportion of HSV-infected people worldwide who have symptomatic disease, as symptoms may be mild or simply not recognized as herpes. In the United States of America, about 15% of people with HSV-2 infection report a prior diagnosis of genital herpes.
When genital herpes symptoms do occur, they take the form of one or more painful genital or anal blisters or ulcers. Herpes symptoms can be treated with antivirals, but after an initial episode, symptoms can recur. Recurrences of genital herpes due to HSV-1 are generally much less frequent than for HSV-2.
Transmission of HSV most often occurs without symptoms. The virus can have a significant negative impact upon an infected person’s mental wellness and personal relationships.
People with orolabial herpes symptoms may face social stigma, and can experience psychological distress as a result. In people with weak immune systems, such as those with advanced HIV infection, HSV-1 can have more severe symptoms and more frequent recurrences. Rarely, HSV-1 infection can also lead to more serious complications such as encephalitis or ocular disease.
WHO is currently working on the development of a global health sector strategy for sexually transmitted infections (STIs), including for HSV-1 and HSV-2, to be finalized for consideration at the 69th World Health Assembly in 2016.
 
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